Hyperpolarization and repolarization both describe increased electronegativity within the cell . 超極化及再極化都表明細胞內負電性的增強。
The hyperpolarization of membrane potential is referred to as the inhibitory junction potential ( ijp ) 這種超極化被稱為抑制性接點電位( inhibitoryjunctionpotential , ijp ) 。
Application of tea could block the outward current , and decrease the mean amplitudes of the initial and second phases of ach - induced hyperpolarization 實驗結果提示ach對與ec共培養的smc影響可能與ec膜上的kc 。
Previous evidence showed that an increase in [ ca2 + ] cyt has been shown to be an early event in the aba signal transduction pathway , and ca2 + channels at the plasma membrane of stomatal guard cells are activated by hyperpolarization and aba Assmann已提出較為完善的氣孔保衛細胞信號轉導模式,但是對于aba信號轉導的一些中間環節仍是人們不斷探討的問題。
In addition , in the later of 80 ' s , the results obtained in weston and suzuki ' s laboratories and other laboratories showed that a new substance , christened edhf , could be released by ach or other vasoactive substances such as substance p . and the edhf could cause hyperpolarization of the vascular smooth muscle cells in an endothelium - dependent manner 除edrf外, 80年代末weston和suzuki等發現ach等血管活性物質還可使血管內皮釋放一種可使smc膜電位超極化的因子( edhf ) 。 edhf介導的血管舒張不受血紅蛋白和亞甲藍的影響,說明其作用機制與no無關。