Hyperpolarization and repolarization both describe increased electronegativity within the cell . 超極化及再極化都表明細(xì)胞內(nèi)負(fù)電性的增強(qiáng)。
The hyperpolarization of membrane potential is referred to as the inhibitory junction potential ( ijp ) 這種超極化被稱為抑制性接點(diǎn)電位( inhibitoryjunctionpotential , ijp ) 。
Application of tea could block the outward current , and decrease the mean amplitudes of the initial and second phases of ach - induced hyperpolarization 實(shí)驗(yàn)結(jié)果提示ach對(duì)與ec共培養(yǎng)的smc影響可能與ec膜上的kc 。
Previous evidence showed that an increase in [ ca2 + ] cyt has been shown to be an early event in the aba signal transduction pathway , and ca2 + channels at the plasma membrane of stomatal guard cells are activated by hyperpolarization and aba Assmann已提出較為完善的氣孔保衛(wèi)細(xì)胞信號(hào)轉(zhuǎn)導(dǎo)模式,但是對(duì)于aba信號(hào)轉(zhuǎn)導(dǎo)的一些中間環(huán)節(jié)仍是人們不斷探討的問(wèn)題。
In addition , in the later of 80 ' s , the results obtained in weston and suzuki ' s laboratories and other laboratories showed that a new substance , christened edhf , could be released by ach or other vasoactive substances such as substance p . and the edhf could cause hyperpolarization of the vascular smooth muscle cells in an endothelium - dependent manner 除edrf外, 80年代末weston和suzuki等發(fā)現(xiàn)ach等血管活性物質(zhì)還可使血管內(nèi)皮釋放一種可使smc膜電位超極化的因子( edhf ) 。 edhf介導(dǎo)的血管舒張不受血紅蛋白和亞甲藍(lán)的影響,說(shuō)明其作用機(jī)制與no無(wú)關(guān)。
The objectives of this study were : ( 1 ) to establish a model of co - culture of endothelial and smooth muscle cells ; ( 2 ) - 5 to ascertain the effects of the ach on the membrane potential of the rabbit aortic smooth muscle cells and bovine aortic endothelial cells ; ( 3 ) to clarify the role of gap junctions in ach - induced endothelium - derived hyperpolarization in rabbit aortic smooth muscle cells ; ( 4 ) to examine the contribution of both no and gap junction to the ach - induced endothelium - derived hyperpolarization in rabbit aortic smooth muscle cells Edhf的性質(zhì)尚未確定,有的研究認(rèn)為其與ec的k ~ +通道有關(guān),而有的研究認(rèn)為平滑肌的內(nèi)皮源性超極化與ec和smc之間的縫隙連接( gapjunction )有關(guān),因?yàn)閮烧咄ㄟ^(guò)縫隙連接有著電偶聯(lián)作用。本文利用細(xì)胞共培養(yǎng)和膜片鉗技術(shù),探討縫隙連接在血管張力調(diào)節(jié)中的作用。